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What Baldness Is and Why It Develops
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(Presented herein information taken from several chapters in the newly published 225-page paperback book,Bald No More, Preventing and Successfully treating Hair Loss for Both Men and Women by Morton Walker, DPM, issued by the Kensington Publishing Corporation.
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At age twenty-six, Phillip Morlork of Toronto, a printer"s apprentice, went through a personal period of emotional devastation when Sarah Valentine, the girl whom he was proposing to marry, broke off their relationship. The tragedy happened simply because it appeared that Phillip was going to look like his very bald father. In contrast, this ex-girlfrend's own dad possessed hair as thick and tight as a Berber rug, and she was much admiring of her pater.
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Because their meetings had been mostly at night in parked cars, darkened taverns, poorly lit social gatherings or in dim restaurants, Sarah had not noticed that the top of Phillip's scalp held a small pancake-size patch of bareness. One evening, before he took the opportunity to propose marriage, Sarah ran her fingers through Phil's hair and stopped in midstroke. She had found the bald spot. Directly after that she dropped him, and, unfortunately, was honest enough to tell the heart-broken fellow why. Looking to the future, Sarah had said, "I just can't see myself with a bald boyfriend." Little did she know that it had been his intention to make himself her bald husband.
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The Long and Short of Hair Growth
In the fairy tale, when Rapunzel let down her hair from the tower in which she was imprisoned, for the prince to climb up, she had no clue that a genetic defect probably lay at the root of her golden locks. But a 1994 experiment in developmental biology conducted as the University of California, San Francisco, revealed that the presence of a growth factor contrary to its name - limits the length that hair will grow. "When the factor is missing, hair grows very, very long," reported Gail L. Martin, PhD, a biologist working at that webt coast university.
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Along with other researchers, Dr. Martin was studying a chemical messenger called fibroblast growth factor 5 in mice that lacked the gene for this particular messenger. The scientists were observing how its loss would affect embryonic development. To their surprise, the newborn mice looked and acted normal - at least at first. But a few weeks after their birth, she and her colleagues noticed that the young mice with missing growth factor looked a bit shaggy. Their hair grew longer than what's usual far baby mice.
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Normally, hair grows in cycles. First, a hair follicle develops. Deep inside it lies a bud of mesodermal tissue which causes the bud to divide and sprout as a hair. Eventually, as expected, it stops growing. The follicle becomes quiescent, and the hair eventually falls out so that the cycle then begins again.
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The Martin research group's genetic analyses indicate that a known abnormal factor called the angora gene is actually a variant of the gene for this growth factor. And Dr. Martin says that people, too, may have the angora variant. According to her concept, fibroblast growth factor 5 is the first, but probably not the only, chemical signal discovered for the hair cycle. "There's obviously a backup signal, because the hair doesn't grow forever," she noted during her presentation to the annual meeting of the Society for Cell Biology, that was held in San Francisco.
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There is no greater right-between-the-eyes reminder that your youth is checking out and leaving no forwarding address than the first time you see a drain clogged with your own hair. So many different forms of baldness exist which could require defensive actions that their number may seem overwhelming.
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The Numerous Forms of Baldness
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Baldness, referred to by the medical profession as alopecia,is the partial or complete loss of hair from the head or the body or both. It may result from a genetic trait, systemic disease, hormonal defect, drug side effect, aging, anticancer treatment, skin disorder, another systemic source, or from some local cause. Here are examples of the more common baldness conditions:
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In alopecias areata, there are well-defined bald patches, often round or oval in shape. They present themselves on the head, beard, and other hairy parts of the body. Even if the condition clears up within a year without treatment, it's common for the suddenly visible alopecia areata to recur somewhat later. A few other less common names are used to identify this problem too. It's variably called alopecia celsi, alopecia circumscripta, and Jonston's alopecia.
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Quite often the alopecia areata condition responds well to the hair revitalizing ingredient in thymus gland extract from the calf, which reactivates dormant hair follicles in men and women, revitalizes normal hair cells for fuller, thicker, healthier hair, and is applicable with positive results in almost all cases of thinning hair for both sexes. It's a full program of treatment which, when steadily applied, prevents the alopecia areata from returning.
Alopecia universalis is a complete loss of hair that shows an all parts of the body. It sometimes occurs as an extension of generalized alopecia areata, and the thymus gland extract frequently does work well to correct this condition.
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In patchy alopecia (although "patchy" is nothing more than a descriptive term), areas develop on the parietal (front) and occipital (back) regions of the scalp that look moth-eaten. The condition is suspected of being connected with the invasion of some microorganism inasmuch as such hair loss has occasionally been a secondary characteristic of various infections. Dr. Klio Moessler, one of the main dermatologists at the Dermatological Department of the Municipal Clinics of Darmstadt in Germany, who participated in research on diseases producing baldness and new potential hair-growing products, points out, "Patchy alopecia may come from a fungal or bacterial infection or from genetic defects involving the hair. It occurs in cicatricial (scar forming) alopecia, alopecia areata, and some skin diseases." Thymus gland extract may not be effective in treating some of the more complicated causes of patchy alopecia.
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For alopecia totalis (complete baldness), all the hair on the scalp is lost. This is an uncommon head hair defect with no known cause, but it does respond to the calf thymus preparation referred to.
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With the three types of alopecia areatas (patchy, universalis, and totalis) evidence is mounting that an immunological signal is involved. In the double condition diagnosed as alopecia areata totalis et universalis the entire head and body of an individual becomes bald. Hair disappears from the pubic region, armpits, eyelashes, eyebrows, chest, legs, beard, and other areas. It has been proven, in clinical studies that thymus gland extract is useful in reversing the effects of the combination condition of alopecia areata totalis et universalis as well.
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You probably are aware that baldness has been considered irreversible and there hasn't been any corrective treatment before today. Immunological aspects of alopecia newly discovered have changed all that.
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In alopecaa disseminate, also referred to as alopecia diffusa, there is hair loss around the whole scalp or even from other parts of the body: The cause may be a nutritional deficiency (especially lack of zinc or iron), a dysfunction of the thyroid gland, a polluting intoxicant, or some chronic and generalized illness. Alopecia diffusa can't be corrected with thymus gland extract applied topically unless the underlying difficulty is found and eliminated.
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As regarding alopecia androgenetica (also known in dermatology as alopecia hereditaria), approximately half of the adult males residing in the United States and other Western industrialized countries exhibit this condition. Dr.Moessler told me that at least 65% of all German men suffer from the problem. In men, some of the other names for the condition of alopceia androgenetica are male pattern baldness, androgenetic alopecia, premature baldness, seborrheic alopecia, common baldness, hereditary baldness. In women, alopecia androgenetica is referred to as female pattern baldness or diffuse alopecia.
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Rodney Dauber, MA, MB, ChB, who is consulting dermatologist at the Churchill Hospital in Oxford and clinical senior lecturer in dermatology at Oxford University, both in the United Kingdom, and Dominique Van Neste, MD, PhD, Director of the Skin Study Center in Tournai, Belgium, have reported: "Androgenetic alopecia probably occurs to a degree in all adults some time after puberty - only being obvious in some women in old age."
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The lose of hair is strongly suspected by dermatologists both in the United States and in Europe, to arise from a baldness gene. For both men and/or women with androgenetic alopecia, this gene is suspected to be inherited from their fathers and occasionally from their mothers.
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The Conjectured Reason for Androgenetic Alopecia
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But with all the conjecture among hair specialists, the true reason for alopecia androgenetica to appear is not entirely known. Dermatologists do recognize that it is the most common form of baldness showing up in males and females. Its onset occurs at puberty in genetically predisposed individuals, and the condition is an autosomal dominant disorder. (Autosomal dominant means that alopecia androgenetica has a pattern of inheritance in which a dominant gene on a nonsex determining chromosome [the autosome] makes a certain characteristic of baldness. Affected individuals usually have a bald parent. However, normal children of the affected parent do not carry the baldness trait. Thus, among two male siblings having a bald father, one son may be bald and the other not. The nonbald brother will not pass on the chromosome for baldness to his sons or daughters, but the bald brother may do so.
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Idiopathic male / female pattern baldness is a separate condition, too. The term, idiopathic, merely means that the medical profession acknowledges that it has not determined the cause of this form of baldness.
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To clarify hereditary baldness, in alopecia androgenetica or alopecia hereditaria, the male pattern baldness and female pattern forms result from sex-influenced dominant inheritance. Androgen (meaning hormonal) stimulation is required to produce hair loss in heterozygous individuals (in which there are two different genes situated at the same place on matched chromosomes). For example, the individual with male pattern baldness could have inherited the bald-headed gene from one parent (mother or father) and the alternative gene from the other parent. The offspring (boy or girl) of a heterozygous carrier of the bald-headed gene has a 50% chance of inheriting this gene from his or her parent. There is a relationship of androgenetic alopecia and increased circulating androgens, at puberty, which probably represents one of the precipitating events in such a heterogeneous hair disorder.
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Furthermore, the cause of androgenetic alopecia appears to be related mostly to androgen metabolism in the skin, the hair follicle, and the sebaceous gland lobule. Hair scientists have found hormonal abnormalities of cytosol and nuclear cell receptors and cytoplasmic-carrying proteins and minerals especially within the body's metabolism of calcium and irons.
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More Basic Underlying Sources of Baldness
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Biology and body chemistry gone awry simultaneously are the usual causes of baldness, particularly in men but also in those women who may possess too much testosterone - the male hormone. In that case, a woman could be exhibiting female pattern baldness (FPB) by her hair's obvious thinning as a result of chronic fallout caused by an excess of male hormone being generated by her endocrine system.
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Male pattern baldness most likely starts with testosterone, the hormone produced in the testes that helps make a man a man (and go bald like a man). Testosterone is a naturally occurring hormone that stimulates the growth of male (androgen) characteristics. As testosterone flows through the body, it interacts with an enzyme called 5-alpha reductase, which is concentrated in the genitals and skin. The enzyme converts testosterone into a more potent hormone called dihydrotestosterone (DHT), which some urologists have referred to as "testosterone times ten.." It is formed directly from testosterone in tissue, particularly in the secondary sex organs and is higly biologically active, except in muscle and bone. DHT is necessary for male external genital development, such as the formation of the penis, scrotum, and proatate.
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In the scalp, each hair follicle is genetically programmed to react differently to DHT. Men with MPB have DHT sensitive follicles at the front or top of their heads, which wither and die from extended exposure to the hormone. It stands to reason that the onset of balding might be prevented biologically by one of several strategies:
A. By stopping the body's production of testosterone.
B. By stopping the testosterone from becoming DHT
C. By blocking DHT before it gets to follicles.
D. By making follicles less sensitive to DHT
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A man interested in curing his baldness would probably skip strategy A, because it involves castration at an early age. Eunuchs never go bald. Although the German scientists who have perfected the hair-growing qualities of the calf-derived thymus product to which I have alluded don't exactly know how it works, they believe it does accomplish at least one of the remaining B, C, or D strategies, or all three, or just two of the three. Perhaps strategy D offers the most logic relating to the product's action: making follicles less vulnerable to hormonal ravages.
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Manfred Hagedorn, MD, chief of Dermatology at the Municipal Clinics of Darmstadt, points out that baldness has been shown under the microscope to be an auto-immune disease wherein one's leucocytes consisting of lymphocytes and macrophages, actually attack hair follicles and cause them to go into dormancy.
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In alopecia androgenica, another separate disease, a male type baldness associated with excessive endocrine gland (androgenic) activity prevails in women as well. This specifically female condition labelled FPB (female pattern baldness) is similar to alopecia androgenetica. It has a genetic background coming under the influence of androgen hormones in which the woman possesses more testosterone than she needs.
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In Chapter Thirteen of my book, Bald No More, is the frst clinical study conducted by Prof. Dr. Hagedorn and Dr. Moessler who utilized the new German hair-growing product and produced a reversal of alopecia androgenetica in 67% of men and 100% of women participating in their investigation. Also the two dermatologists carried out a second clinical study that demonstrated the regrowth of hair from the application of this thymus gland extract product from the calf for 43% of participating bald men and 94% of bald women.
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Confused by Baldness Terminology?
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Among the striking anatomical features of human hair are its distribution, its variety, and its sparsity compared with that of other primates. Human hair is not vestigial; it varies in type, density, and length. The hair strands and their follicles have a unique anatomy unlike any other structures in or on the human body.
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The lines of differentiation in internal medicine, plastic surgery, and dermatology regarding differences in these various baldness conditions are not clearcut. So if you are confused by all of the descriptive hair loss definitions, you are not alone. Many physicians have their own problems with diagnosing the reason for scalp disorders. As an example of massive confusion, the medical textbook, Disorders of Hair Growth: Diagnosis and Treatment, distinguishes among several hundred types of individual hair losses occurring from diseases, disorders, or deficiencies. Here is a list of just a few of them taken from, a second dermatology textbook.
Lupus erythematosus Childbirth, postpartum period
Lichen planes pilaria Episodes of fever
Planopilaris Surgery
Fseudopelade Drugs
Sclaroderma Endocrinopathias e.g. excess androgen
Bullbous pemphigoid Nutritional metabolic disorders
Epidermolysie bullosa acquista Autoimmune diseases
Folliculitis secondary to infection Chemical and physical damage
Granulomatous inflammation Nonscarring pseudopelade
Benign neoplasms Malignant tumors
Ichthyosis congenita Hair traction from styling
Seborrbeic dermatitis Follicular mucinosis

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During my appearance as the guest on a radio talk show in Vancouver, British Columbia, Canada I was discussing hair loss. A program caller quizzed me and then stated, "I know why men go bald. They wear hats that cut off their blood circulation and prevent the scalp from 'breathing."' This caller's comment was sheer nonsence. There are literally hundreds of reasons for baldness and wearing a hat is not one of them.
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At least 37 additional types of alopecia could be mentioned, but many of them are so very rare they needn't be defined, unless we come across them in discussing treated patients. For instance, in Chapters Seven and Eight of my book, I describe successful prevention and immediate reversal of alopecia medicamentosa, diffuse hair loss, most notably froze the scalp, caused by the administration of cytotoxic chemotherapy for cancer.
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During several months when my wife Joan was taking elevated does of the mineral germanium in the form of encapsulated GE 132 powder as part of her cancer therapy protocol, she underwent hair loss. While ordinarily a trace mineral taken as a nutritional supplement in tiny doses; if consumed in massive amounts to kill cancer cells, GE 132 acts like a cytotoxic agent used in chemotherapy. This high dosage germanium was part of Klinik Winnerhof's anticancer treatment program as prescribed by Helmut G. Keller, M.D. At one point, Joan, was losing between 600 and 1000 hair strands per day.
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Then, I called upon the hair-preserving company That distribute thymus gland extract, ThymuSkin®, the most effective preparation known for the treatment of hair loss due to chemotherapy, alopecia areata, or patterned baldness. It is free of any type of adverse side effects and immediately stops hair from falling out. Within just a few days of using ThymuSkin®Shampoo, Treatment solution, and Hair Gel, my wife's hair loss discontinued. Her bald spots are now filling in too.
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What Thymus Gland Extract Does and How It Is Supplied
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There are five different thymus gland extracts under the umbrella name of ThymuSkin® hair products. All of them preserve hair follicles and/or stimulate dormant but live follicles into regrowth. Basically, a consumer can make do with only two products to start the program.
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Being the two items investigated during clinical studies done in Germany, theThymuSkin® Shampoo and ThymuSkin® Hair Treatment Lotion are the really important formulations to use. The fundamental principle for the shampoo is for cleansing the scalp with tiny penetrating thymus peptides. Since these peptides are broken down into the smallest of sizes - only 300 angstroms in length - they do diffuse themselves into the hair follicles better than any other mode of cleanser. They clean out each follicle of accumulated oil, dirt, debris, and other waste and prepare the scalp for receiving the treatment lotion.
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These two products - shampoo and hair treatment lotion - work synergistically, and they are packaged together in kits - small and large. The small kit contains one bottle of 100 milliliters (ml) of shampoo and one bottle of 100 ml of the treatment lotion. (A milliliter is one thousandth of a liter, and one liter is 33.81.4 ounces [ozs]. For ease of figuring, you may consider that one oz contains 30 ml, and there are 30 drops in one ml.) Each of the small-size kit products will last six to eight weeks, depending on the volume of hair to which they are applied. Also, a larger area of scalp that is thinning or balding actually uses less liquid than hairy areas because the solutions are so much more readily spread across the scalp's smooth skin.
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The larger ThymuSkin®; hair preservation kit contains one 200 ml bottle of shampoo and one 200 ml bottle of hair treatment lotion. Each of them lasts approximately there to four months, again depending on the volume of hair and the areas of thinning or balding.
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The usual experience of users is that large amounts of hair fallout around the sink or in the shower as happend to my wife, diminish steadily. The user notices a decrease of hair fall within the first week of only using the shampoo. If you, as the user, have observed balding or thinning of head hair for less than five years, you are likely to notice new hair growth as early as two to three months. If your hair loss has been occurring far longer than five years, than it could take anywhere from twelve to eighteen months for you to see new hair growth. Usually after eighteen months, if the ThymuSkin® combination of ingredients hasn't helped, you are proving that the hair follicles on remaining bald areas are dead and nothing applied will help to restore them to life.
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The ThymuSkin® Hair Treatment Gelis a higher concentractions of thymus peptides, vitamins, minerals, enzymes, and amino acids. It was developed to be used in conjunction with the shampoo arid treatment lotion. The gel is a kind of "miracle grow" that tends to produce follicular stimulation more quickly. Because it's so very concentrated, only place the gel onto the balding or thinning areas for intervals of twelve hours. And use it separately from the shampoo and lotion. That is, you might apply the gel in the evening and go to sleep with it spread over the scalp; then in the morning shampoo it away and put on the lotion for the balance of your working day.
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The ThymuSkin® Hair Treatment Mask is more of a setting lotion. It is prepared as a gelled type of substance that one applies to towel-dried hair after the shampoo and hair treatment lotion have been used. The hair mask is placed between the hairs and combed through so that the hairs become covered with a protective coating. This coating holds off ultraviolet rays, X-rays, cosmic rays, and other adverse environmental influences from affecting new hair growth. Balding airline personnel who fly as part of their occupation would benefit greatly from usage of the hair treatment mask. Also it makes the hair feel healthy to the touch and looking shiney.
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The ThymuSkin® Conditioner Creme Rinse is somewhat more expensive than other hair rinses, but it makes the hair strands so very soft and silky to the touch This terture holds in place for an extra long period. The conditioner contains the thymus peptides and avocado oils which help to regulate the fat metabolism of the scalp. It may be left in place to condition for up to fifteen minutes (with a towel wrapped around the head) twice a week, without worry of clogging the hair follicles.
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If one wishes to condition the hair with something other than the ThymuSkin® Conditioner Creme Rinse, try to acquire a natural cosmetic product and only put it on the hair ends. Otherwise, if you allow this substitute coditioner to coat the scalp, a problem arises because the ThymuSkin® Shampoo has already cleaned the scalp and its hair follicles. The other conditioner will coat the scalp, and any purpose of cleansing the scalp will be defeated.
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As for other shampoos; they may posses peptides which break- down to the larger size of 500 angstroms, That's usual. These bigger peptides won't penetrate the scalp to clean out the hair follicles. Clogging from debris then develops.
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Perms, dyeing. and highlighting for hair have no adverse effect on the success of treating withThymuSkin.® However, it's recommended that you should use high-quality branded products for these purposes. And afterward, neutralize possible abuse of your hair follicles by reapplying theThymuSkin® Conditioner Creme Rinse and Hair Treatment Mask.
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Using a hair dryer is not recommended for it causes the scalp to perspire, become dehydrated, and undergo loss of necessary minerals. Such an environmental response of the scalp eventually results in skin dryness, itching, drandruff, and hair loss. If you cannot dispense with the use of a hair dryer, consider following these practices:
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  • Hold the hot air blowing apparatus to one side of the head and direct it from below in an upward direction and away from the scalp.
  • Insure that no hot air reaches the scalp directly:
  • After applying hair treatment lotion, under no circumstances use a hair dryer or the lotion will be dissipated into the air as an evaporant. Your effort and investment will then be lost.
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How Often to Apply the Hair Treatment
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To insure new hair growth or to prevent any further receding hairline ThymuSkin®Hair Treatment should be applied to the scalp twice a day for four weeks. Thereafter it can be put on once a day for six to eighteen months, depending on the severity of hair loss. After one's baldness is decreased and hair growth is more predominant, usage can first be reduced to every second day and then to twice a week thereafter. When the ThymuSkin® Hair Treatment Revitalizer is applied to the scalp, a brisk one-minute massage should be administered each time. Additionally wash the hair at least twice a week with the complementing ThymuSkin® Shampoo. This is excellent therapy for the hair of people who have been victims of one or more of the four particular scalp problems, female or male patterned baldness, Alopecia medicamentosa (chemotherapy), Alopecia totalis, and Alopecia universalis.
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Only those separate regions, on the scalp containing enough live hair follicles will show good hair growth. If patches of the scalp hold, dead follicles, no hair will arise from them. Usually hair preservation and even new hair growth will happen for people who are still in the process of losing their hair. For those who experienced a sudden baldness occurring from allergy, deficiency, menopause, pregnancy; metallic toxicity, or another reason associated with pathology, the health problem is best corrected fast. Then, without questionThymuSkin® does the job exceedingly well. If this product is applied one week before and during mild or moderate chemotherapy, there won't be any hair loss for the patient. This is verified by numbers of German oncologists.
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Keep in mind that ThymuSkin® is a doctor-recommended cosmetic product sold without prescription, over-the-counter, and for self-administration.
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For longstanding baldies like me, in which male pattern baldness has been an inherited trait (Alopecia hereditaria) - my father and grandfather being bald at relatively young ages - there is no hope of much hair restoration. Even so, Dr. Moessler told me, "If somebody has members of his or her family who are bald, and he or she does not want to be bald as well, he or she should use ThymuSkin®as a preventative, starting at a young age and continuing for a long period."
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In association with the highly penetrating small-chained peptides, the ThymuSkin® nutrients are carried into the blood stream and travel around to the rest of the body. The combination preparations are stimulating the growth of hair and, optionally, the growth of skin and nails, as well. Through the skin of your scalp, the following nutrients, under US Patent PCT/DE95/01745, can nourish your total physiology, most especially your hair follicles, epidermis, and nails:

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Provitamin A (beta carotene) Methionine
Vitamin B (thiamin) Cystine
Vitamin B1 (riboflavin) Flax oil
Pantothenic acid Lactalbumin
Biotin Calcium
Folic acid (folate) Glycerol
Nicotinamide Vegetable oils
Vitamin B (pyridoxine) CoEnzyme Q (ubiquinone)
Vitamin B6 (cyanocobalamine) Stabilizer E 420
Vitamin C (ascorbic acid) Selenium
Vitamin E (DL alpha tocopherol) Calf thymus extract
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